MESSAGE FROM THE CHIEF
Hobart W. Harris, M.D., M.P.H.
Professor of Surgery
Chief, Division of General Surgery
Kimberly Kirkwood, M.D.
Associate Professor of Surgery
Contact Information
Clinical Telephone: (415) 353-2161
Academic Telephone: (415) 476-0762
Fax: (415) 476-8694
kirkwoodk@surgery.ucsf.edu
Education
- B.A., Reed College, 1981
- M.D., University of Pennsylvania, 1985
Residencies
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Massachusetts General Hospital, Resident, Surgery, 1986-88
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Massachusetts General Hospital, Resident, Surgery, 1991-92
- Massachusetts General Hospital, Chief Resident, Surgery, 1992-93
Fellowships
- University of California, Los Angeles, Postdoctoral Fellow, Gastrointestinal Physiology, 1988-91
Board Certification
- American Board of Surgery, 1994
Clinical Expertise
- Pancreatic Cancer and Benign PancreaticTumors
- Biliary Tract Surgery
- Gallbladder Surgery
- Stomach and Gastrointestinal Surgery
Biography
Dr. Kimberly Kirkwood specializes in the treatment of pancreatic cancer as well as tumors of the stomach, spleen, gallbladder, bile duct and gastrointestinal tract. A skillful laparoscopic surgeon, she is an expert in applying minimally invasive techniques to treat abdominal tumors and has a particular interest in improving outcomes for patients after pancreas surgery. Dr. Kirkwood is a member of the UCSF Helen Diller Family Comprehensive Cancer Center and Participates in Cancer Center Clinical Trials.
Dr. Kirkwood earned a medical degree at the University of Pennsylvania and completed a surgical residency and a fellowship in advanced general surgery with a focus on pancreatic surgery at Massachusetts General Hospital. She also completed a three-year surgical research fellowship at UCLA in hepatobiliary disease. Dr. Kirkwood is active in many national societies including the Pancreas Club, Society for Surgery of the Alimentary Tract, Association for Academic Surgeons and she is on the Executive Council of the Society of University Surgeons. Her NIH-funded research focuses on pancreatic inflammatory pain and on the many factors which may improve outcomes for patients following resection of pancreatic tumors. She is an associate professor of surgery at UCSF.
Selected Publications
- Lightner AM, Glasgow RE, Jordan TH, Krassner AD, Way LW, Mulvihill SJ, Kirkwood KS. Pancreatic resection in the elderly, J Am Coll Surg, May/2004;198(5):697-706.
- Grahn S, Alvarez J, and Kirkwood KS. Trends in Laparoscopic Splenectomy for Massive Splenomegaly. Archives of Surgery, 2006;(141):755-762.
- Lu EJ, Curet MJ, El-Sayed YY, Kirkwood KS. Medical versus surgical management of biliary tract disease in pregnancy. Am J Surg, Dec/2004;188(6):755-9.
- Grady EF, Yoshimi SK, Maa J, Valeroso D, Vartanian RK, Rahim S, Kim EH, Gerart D, Gerard N, Bunnett NW, and Kirkwood KS. Substance P mediates inflammatory edema in acute pancreatitis via activation of the neruokinin-1 receptor in rats and mice. BR J Pharmacol. 130: 505-512, 2000.
- Maa J, Grady EF, Kim EH, Yoshimi SK, Bunnett NW, and Kirkwood KS. Neurokinin-1 receptor desensitization and neutral endopeptidase terminate SP-induced pancreatic inflammation. Am J Physiol Gastroint Liver Physiol. 279: G726-732, 2000.
- Kirkwood KS, Bunnett NW, Maa J, Castagliolo I, Liu B, Gerard N, Zacks J, Pothoulakis C, and Grady EF. Deletion of neutral endoeptidase exacerbates intestinal inflammation induced by clostridium difficile toxin A. Am J Physiol Gastrointest Liver Physiol. 281: G544-G551, 2001.
- Hutter MM, Wick E, Lightner AM, Maa J, Zerega EC, Richmond AC, Jordan TH, Grady EF, Mulvihill SJ, Bunnett NW, and Kirkwood KS. Transient receptor potential vanilloid receptor-1 (TRPV1) Promotes neurogenic inflammation in the pancreas via activation of the neurokinin-1 receptor (NK1-R.). Pancreas. 30: 260-265, 2005.
- Wick EC, Hoge SG, Grahn SW, Kim E, Divino LA, Grady EF, Bunnett NW, and Kirkwood KS. Transient receptor potential vanilloid 1, calcitonin gene-related peptide, and substance P mediate nociception in acute pancreatitis. Am J Physiol Gastrointest Liver Physiol. 290: G959-G969, May/2006.